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JNCI Journal of the National Cancer Institute 2004 96(7):554-555; doi:10.1093/jnci/djh090
© 2004 by Oxford University Press
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© 2004 Oxford University Press

CORRESPONDENCE

RESPONSE: Re: Prostate Carcinogenesis in N-methyl-N-nitrosourea (NMU)–Testosterone-Treated Rats Fed Tomato Powder, Lycopene, or Energy-Restricted Diets

Steven K. Clinton, Thomas M.-W. Boileau, John W. Erdman, Jr.

Affiliations of authors: Division of Hematology and Oncology, Department of Internal Medicine, Arthur G. James Cancer Hospital (SKC); Department of Human Nutrition, The Ohio State University, Columbus, OH (TWMB); Division of Nutritional Sciences, The University of Illinois, Champaign-Urbana (JWE).

Correspondence to: Steven K. Clinton, MD, PhD, Arthur G. James Cancer Hospital, A431 Starling Loving Hall, 320 West 10th Ave., The Ohio State University, Columbus, OH 43210 (e-mail: clinton-1@medctr.osu.edu)

The first 10% of the full text of this article appears below.

The correspondence by Limpens et al. and the editorial by Drs. Gann and Khachik (1) that accompanied our article reinforce the complexities surrounding the relationships among tomato products, lycopene, and prostate carcinogenesis. Both highlight uncertainties regarding dose–response relationships for carotenoids and biologic effects. Indeed, chemists have long understood that carotenoids can be antioxidants under certain conditions and pro-oxidants under other conditions (2,3). Limpens et . . . [Full Text of this Article]


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