© 2004 by Oxford University Press
© 2004 Oxford University Press
CORRESPONDENCE |
Re: Integrin
3 Leu33Pro Homozygosity and Risk of Cancer
Affiliations of authors: Department of Biosciences at Novum, Karolinska Institute, Huddinge, Sweden (QJ, KH, AF); Division of Molecular Genetic Epidemiology, German Cancer Research Center (DKFZ), Heidelberg, Germany (KH, AF); Department of Tumor Biology, Centre of Oncology, Maria Sklodowska-Curie Institute, Gliwice, Poland (EG); Institute of Human Genetics, University of Heidelberg, Heidelberg, Germany (RK); Department of Pathology, Huddinge Hospital, Huddinge (MS).
Correspondence to: Qianren Jin, MD, Department of Biosciences at Novum, Karolinska Institute, SE-141 57, Huddinge, Sweden (e-mail: qianren.jin@cnt.ki.se)
| The first 10% of the full text of this article appears below. |
Altered expression of the
3 integrin gene has been implicated in the metastasis of breast cancer (1). A single-nucleotide polymorphism at codon 33 of the
3 integrin gene that exchanges a leucine (Leu) for a proline (Pro) seems to modify the function of cells expressing
3 integrins [(2) and
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Del.icio.us What's this?
3 Leu33Pro Homozygosity and Risk of Cancer
J Natl Cancer Inst 2004 96: 235.
3 Leu33Pro Homozygosity and Risk of Cancer
J Natl Cancer Inst 2005 97: 778-779.
3 Leu33Pro Homozygosity and Risk of Cancer
J Natl Cancer Inst 2005 97: 779-780.
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