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JNCI Journal of the National Cancer Institute 2004 96(1):75; doi:10.1093/jnci/djh018
© 2004 by Oxford University Press
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© 2004 Oxford University Press

CORRESPONDENCE

Re: Etiology of Pancreatic Cancer, With a Hypothesis Concerning the Role of N-Nitroso Compounds and Excess Gastric Acidity

Gabriele Capurso, Gianfranco Delle Fave, Nick Lemoine

Affiliations of authors: Cancer Research UK Molecular Oncology Unit, Imperial College London Faculty of Medicine at Hammersmith Campus, London, U.K. (GC, NL); Digestive and Liver Disease Unit, II Medical School, University "La Sapienza," Rome, Italy (GC, GDF).

Correspondence to: Gabriele Capurso, MD, Cancer Research UK Molecular Oncology Unit, Imperial College London Faculty of Medicine at Hammersmith Campus, Du Cane Rd., W12 0NN London, U.K. (e-mail: gabriele.capurso@cancer.org.uk)

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We read with great interest the exhaustive review from Risch (1), which focused on the possible role of gastric acid hypersecretion in the etiology of pancreatic cancer. The author presented several strands of evidence connecting alterations of gastric physiology in the course of Helicobacter pylori infection with pancreatic cancer. It is hypothesized that the decrease in . . . [Full Text of this Article]


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Correspondence about this Article

RESPONSE Re: Etiology of Pancreatic Cancer, With a Hypothesis Concerning the Role of N-Nitroso Compounds and Excess Gastric Acidity
Harvey A. Risch
J Natl Cancer Inst 2004 96: 75-76. [Extract] [Full Text] [PDF]