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JNCI Journal of the National Cancer Institute 2003 95(7):559-560; doi:10.1093/jnci/95.7.559
© 2003 by Oxford University Press
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Journal of the National Cancer Institute, Vol. 95, No. 7, 559-560, April 2, 2003
© 2003 Oxford University Press

CORRESPONDENCE

Re: Hormonal Markers and Hepatitis B Virus-Related Hepatocellular Carcinoma Risk: A Nested Case–Control Study Among Men

Ying-Man Sung, Nelson Leung-Sang Tang, Paul Bo-San Lai, Paul Kay-Sheung Chan, Francis Ka-Leung Chan

Affiliation of authors: Y.-M. Sung, N. L.-S. Tang (Department of Chemical Pathology), P. B.-S. Lai (Department of Surgery), P. K.-S. Chan (Department of Microbiology), F. K.-L. Chan (Department of Medicine and Therapeutics, Faculty of Medicine), The Chinese University of Hong Kong, Shatin, Hong Kong, China.

Correspondence to: Nelson Leung-Sang Tang, M.D., F.R.C.P.A., Department of Chemical Pathology, Faculty of Medicine, The Chinese University of Hong Kong, Shatin, Hong Kong, China (e-mail: nelsontang@cuhk.edu.hk).

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Steroid 5{alpha}-reductase type II (SRD5A2) catalyzes the conversion of testosterone to a more potent androgen, dihydrotestosterone, and plays an important role in steroidogenesis. One hypothesis for a hormonal etiology in hepatocellular carcinoma (HCC) was raised by a study that showed a positive association between testosterone levels and . . . [Full Text of this Article]


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Am. J. Epidemiol., April 15, 2009; 169(8): 990 - 995.
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