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JNCI Journal of the National Cancer Institute 2003 95(13):1008-1009; doi:10.1093/jnci/95.13.1008
© 2003 by Oxford University Press
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Journal of the National Cancer Institute, Vol. 95, No. 13, 1008-1009, July 2, 2003
© 2003 Oxford University Press

CORRESPONDENCE

Re: Discrete Alterations in the BZLF1 Promoter in Tumor and Non-Tumor-Associated Epstein–Barr Virus

Joanna H. M. Tong, Kwok Wai Lo, Florence W. L. Au, Dolly P. Huang, Ka-Fai To

Affiliation of authors: Department of Anatomical and Cellular Pathology, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong.

Correspondence to: Ka-Fai To, F.R.C.P.A., Department of Anatomical and Cellular Pathology, Prince of Wales Hospital, The Chinese University of Hong Kong, Shatin, NT Hong Kong (e-mail: kfto@cuhk.edu.hk).

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In their recent article, Gutiérrez et al. (1) identified three Epstein–Barr virus (EBV) variants in the BZLF1 promoter Zp (Zp-P [considered the prototypical sequence], Zp-V3, and Zp-V4) and postulated that these might result in different lytic potentials and be differentially expressed among individuals with nonmalignant and malignant diseases. Hong Kong and Southern China are considered to be endemic . . . [Full Text of this Article]


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