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JNCI Journal of the National Cancer Institute 2002 94(10):703; doi:10.1093/jnci/94.10.703-a
© 2002 by Oxford University Press
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Journal of the National Cancer Institute, Vol. 94, No. 10, 703, May 15, 2002
© 2002 Oxford University Press

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ICAM-1 Null Mutation and Pulmonary Fibrosis

The efficacy of radiation therapy for lung cancer is limited because it induces pulmonary inflammation and interstitial fibrosis. Radiation-induced pulmonary inflammation is mediated by expression of intercellular adhesion molecule 1 (ICAM-1) on endothelial cells of the microvasculature. Hallahan et al. (p. 733) investigated the relationship between radiation-induced inflammation and fibrosis by studying the lungs of irradiated mice that contained or lacked the gene for ICAM-1. The authors found that the lungs of irradiated ICAM-1-deficient mice had fewer infiltrating inflammatory cells than did those of mice bearing . . . [Full Text of this Article]

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