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JNCI Journal of the National Cancer Institute 1999 91(6):557; doi:10.1093/jnci/91.6.557
© 1999 by Oxford University Press
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Journal of the National Cancer Institute, Vol. 91, No. 6, 557, March 17, 1999
© 1999 Oxford University Press

BRIEF COMMUNICATION

p53 and Genetic Susceptibility to Cervical Cancer

Yukio Sonoda, Patricia E. Saigo, Jeff Boyd

Affiliation of authors: Gynecology and Breast Research Laboratory, Departments of Surgery, Pathology, and Human Genetics, Memorial Sloan-Kettering Cancer Center, New York, NY.

Correspondence to: Jeff Boyd, Ph.D., Department of Surgery, Box 201, Memorial Sloan-Kettering Cancer Center, 1275 York Ave., New York, NY 10021.

 In a recent report, Storey et al. (1) provide compelling experimental evidence that two common polymorphic variants of the p53 tumor suppressor protein differ in their susceptibility to degradation mediated by the E6 oncoprotein of human papillomavirus (HPV). Specifically, the p53 protein with arginine at codon 72 was shown to be more susceptible to E6-induced degradation in . . . [Full Text of this Article]

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