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JNCI Journal of the National Cancer Institute 1999 91(21):1888-1889; doi:10.1093/jnci/91.21.1888
© 1999 by Oxford University Press
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Journal of the National Cancer Institute, Vol. 91, No. 21, 1888-1889, November 3, 1999
© 1999 Oxford University Press

BRIEF COMMUNICATION

Prevalence of Human Herpesvirus 8 Infection Before the Acquired Immunodeficiency Disease Syndrome-Related Epidemic of Kaposi's Sarcoma in East Africa

Guy de-Thé, Giovanna Bestetti, Monique van Beveren, Antoine Gessain

Affiliation of authors: Unité d'Epidémiologie des Virus Oncogènes et d'Oncologie Virale, Département des Rétrovirus et CNRS/URA 1930, Institut Pasteur, Paris, France.

Correspondence to: Guy de-Thé, M.D., Ph.D., Unités d'Epidémiologie des Virus Oncogènes et d'Oncologie Virale, Département des Rétrovirus et CNRS/URA 1930, Institut Pasteur, 28 rue du Dr. Roux, 75724, Paris, France (e-mail: dethe@pasteur.fr).

The etiologic role of the recently discovered Kaposi's sarcoma (KS)-associated herpesvirus (KSHV), also called human herpesvirus 8 (HHV8), in the development of all types of KS is now generally accepted (1,2). Long before the epidemic of the acquired immunodeficiency disease syndrome (AIDS) began, East Africa was known as a high-risk area for KS. For example, during the quinquennium 1964 through 1968, the annual incidence of KS in Uganda was 14.6 per million in males, representing approximately 4.6% of the pathologically examined tumors (3). By comparison, Wabinga et al. (4) observed that the annual incidence of KS in Uganda in 1993 was approximately 300 per million in males, representing . . . [Full Text of this Article]

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