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JNCI Journal of the National Cancer Institute 1999 91(19):1685; doi:10.1093/jnci/91.19.1685
© 1999 by Oxford University Press
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Journal of the National Cancer Institute, Vol. 91, No. 19, 1685, October 6, 1999
© 1999 Oxford University Press

CORRESPONDENCE

Re: Cyclin D1 Proteolysis: a Retinoid Chemoprevention Signal in Normal, Immortalized, and Transformed Human Bronchial Epithelial Cells

Herbert Yu

Correspondence to: Herbert (He) Yu, M.D., Ph.D., Section of Cancer Prevention and Control, Feist-Weiller Cancer Center, Louisiana State University Medical Center, 1501 Kings Highway, P.O. Box 33932, Shreveport, LA 71104 (e-mail: hyu@lsumc.edu).

Retinoids are potent antiproliferative molecules with potential implications in cancer prevention. The report by Boyle et al. (1) provides new insights into the mechanism of retinoid-mediated suppression of cell proliferation. The experiments demonstrated that retinoids could reduce cyclin D1 levels in bronchial epithelial cells and that the effect . . . [Full Text of this Article]

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