Skip Navigation

JNCI Journal of the National Cancer Institute 2006 98(22):1665; doi:10.1093/jnci/djj453
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Request Permissions
Google Scholar
Right arrow Articles by Zecevic, M.
Right arrow Articles by Frazier, M. L.
Right arrow Search for Related Content
PubMed
Right arrow Articles by Zecevic, M.
Right arrow Articles by Frazier, M. L.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author 2006. Published by Oxford University Press.

CORRESPONDENCE

RESPONSE: Re: IGF-1 Gene Polymorphism and Risk for Hereditary Nonpolyposis Colorectal Cancer

Maja Zecevic, Christopher I. Amos, Marsha L. Frazier

Affiliations of authors: The Lancet, New York, NY (MZ); Department of Epidemiology, The University of Texas M. D. Anderson Cancer Center, Houston, TX (CIA, MLF)

Correspondence to: Marsha L. Frazier, PhD, Department of Epidemiology, The University of Texas M. D. Anderson Cancer Center, 1155 Pressler Blvd., Unit 1365, Houston, TX 77030 (e-mail: mlfrazier@mdanderson.org).

The first 10% of the full text of this article appears below.

Hereditary nonpolyposis colorectal cancer (HNPCC) is an autosomal dominant disorder caused by DNA mismatch repair (MMR) gene mutations with hMLH1 and hMSH2 being the most frequently mutated (1). Reeves et al. have genotyped a . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?