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JNCI Journal of the National Cancer Institute 2006 98(2):144-145; doi:10.1093/jnci/djj025
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© The Author 2006. Published by Oxford University Press.

CORRESPONDENCE

Re: MC1R, ASIP, and DNA Repair in Sporadic and Familial Melanoma in a Mediterranean Population

Maria Concetta Fargnoli, Tania Spica, Francesco Sera, Giovanni Pellacani, Alessandra Chiarugi, Stefania Seidenari, Paolo Carli, Sergio Chimenti, Ketty Peris

Affiliations of authors: Department of Dermatology, University of L'Aquila, L'Aquila, Italy (MCF, TS, KP); Molecular and Nutritional Epidemiology Unit, CSPO, Scientific Institute of Tuscany, Florence, Italy (FS); Department of Dermatology, University of Modena and Reggio Emilia, Modena, Italy (GP, SS); Department of Dermatology, University of Florence, Florence, Italy (AC, PC); Department of Dermatology, University of Rome "Tor Vergata", Rome, Italy (SC)

Correspondence to: Ketty Peris, MD, Department of Dermatology, University of L'Aquila, Via Vetoio-Coppito 2, 67100 L'Aquila, Italy (e-mail: peris@univaq.it).

The first 10% of the full text of this article appears below.

We read with great interest the article by Landi et al. (1) reporting the association of melanocortin-1 receptor gene (MC1R) variants with melanoma risk and progression in sporadic and familial melanoma patients from northeastern Italy. They observed a two- to fourfold increase in risk of both sporadic and familial melanoma among individuals carrying MC1R variant alleles compared with those carrying wild-type MC1R.

We investigated whether MC1R variants were associated with the risk of sporadic cutaneous melanoma in a population . . . [Full Text of this Article]


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Response to this Correspondence

RESPONSE: Re: MC1R, ASIP, and DNA Repair in Sporadic and Familial Melanoma in a Mediterranean Population
Maria Teresa Landi, Peter Kanetsky, Alisa Goldstein, and Ruth Pfeiffer
J Natl Cancer Inst 2006 98: 145-146. [Extract] [Full Text] [PDF]



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