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JNCI Journal of the National Cancer Institute 2005 97(4):321-322; doi:10.1093/jnci/dji052
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© 2005 Oxford University Press

CORRESPONDENCE

RESPONSE: Re: Association Between Biallelic and Monoallelic Germline MYH Gene Mutations and Colorectal Cancer Risk

Sean P. Cleary, Marina E. Croitoru, Robert Gryfe, Michael Manno, Michelle Cotterchio, Julia Knight, Steven Gallinger

Affiliations of authors: Samuel Lunenfeld Research Institute (MEC, SPC, MM, JK, RG, SG) and Department of Surgery (RG, SG), Mount Sinai Hospital, Toronto, ON, Canada; Ontario Familial Colorectal Cancer Registry, Cancer Care Ontario, Toronto, ON, Canada (MM, MC, JK, SG)

Correspondence to: Steven Gallinger, MD, MSc, FRCS, Rm. 1225, Mount S inai Hospital, 600 University Ave., Toronto, ON, Canada M5G 1X5 (e-mail: sgallinger@mtsinai.on.ca).

The first 10% of the full text of this article appears below.

Dominant and recessive transmission applies to traits or phenotypes and not to genes themselves. A good example of this terminology is sickle cell disease: homozygous carriers have the disease and heterozygous carriers have the sickle cell trait, a milder phenotype of the disease (1,2). It is, of course, true that published reports to date clearly support a recessive mode of . . . [Full Text of this Article]


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Related Correspondence

Re: Association Between Biallelic and Monoallelic Germline MYH Gene Mutations and Colorectal Cancer Risk
Albert Tenesa, Susan M. Farrington, and Malcolm G. Dunlop
J Natl Cancer Inst 2005 97: 320-321. [Extract] [Full Text] [PDF]