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JNCI Journal of the National Cancer Institute 2005 97(4):320-321; doi:10.1093/jnci/dji051
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© 2005 Oxford University Press

CORRESPONDENCE

Re: Association Between Biallelic and Monoallelic Germline MYH Gene Mutations and Colorectal Cancer Risk

Albert Tenesa, Susan M. Farrington, Malcolm G. Dunlop

Affiliations of authors: Colon Cancer Genetics Group, University of Edinburgh, and MRC Human Genetics Unit, Western General Hospital, Edinburgh

Correspondence to: Albert Tenesa, BSc, School of Molecular and Clinical Medicine, The University of Edinburgh, Colon Cancer Genetics Group, 14th Floor, MRC Human Genetics Unit, Western General Hospital, Crewe Road South, Edinburgh EH4 2XU, UK (email: albert.tenesa@ed.ac.uk).

The first 10% of the full text of this article appears below.

We read with interest the paper by Croitoru et al. (1), in which they examined the role of germline MYH gene mutations in colorectal cancer and proposed an autosomal dominant weakly penetrant mode of inheritance. Previous evidence has suggested a recessive mode of inheritance for colorectal polyposis (2). Hence, the novel proposal . . . [Full Text of this Article]


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Response to this Correspondence

RESPONSE: Re: Association Between Biallelic and Monoallelic Germline MYH Gene Mutations and Colorectal Cancer Risk
Sean P. Cleary, Marina E. Croitoru, Robert Gryfe, Michael Manno, Michelle Cotterchio, Julia Knight, and Steven Gallinger
J Natl Cancer Inst 2005 97: 321-322. [Extract] [Full Text] [PDF]