© 2004 by Oxford University Press
© 2004 Oxford University Press
EDITORIALS |
Common Threads of Mucosa-Associated Lymphoid Tissue Lymphoma Pathogenesis: From Infection to Translocation
Correspondence to: Elaine S. Jaffe, MD, Hematopathology Section, Laboratory of Pathology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, 10 Center Drive, Bldg. 10, Rm. 2N202 MSC-1500, Bethesda, MD 20892-1500 (e-mail: elainejaffe@nih.gov)
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In this issue of the Journal, Ferreri et al. (1) add to the growing list of infectious agents that have been associated with extranodal marginal zone B-cell lymphomas of mucosa-associated lymphoid tissue (MALT lymphomas) with their finding that Chlamydia psittaci infection is associated with ocular adnexal lymphomas. Helicobacter pylori infection was first identified as a risk factor for gastric MALT lymphoma, which is invariably preceded by H. pylori-associated follicular gastritis (2). Subsequently, Isaacson et al. (2) showed that MALT lymphomas are antigen-driven clonal B-cell lymphomas and that eradication of H. pylori with antibiotic therapy could lead to lymphoma regression, at least in its early stages, prior to additional genetic events (3). More recently, Borrelia burgdorferi and Campylobacter jejuni have been linked to
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