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JNCI Journal of the National Cancer Institute 2004 96(1):75-76; doi:10.1093/jnci/djh019
© 2004 by Oxford University Press
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© 2004 Oxford University Press

CORRESPONDENCE

RESPONSE Re: Etiology of Pancreatic Cancer, With a Hypothesis Concerning the Role of N-Nitroso Compounds and Excess Gastric Acidity

Harvey A. Risch

Harvey A. Risch, MD, PhD, Department of Epidemiology and Public Health, Yale University School of Medicine, 60 College St., POB 208034, New Haven, CT 06520–8034 (e-mail: harvey.risch@yale.edu)

The first 10% of the full text of this article appears below.

I thank Dr. Capurso and colleagues for their observations concerning the etiologic hypotheses I presented in my review of gastric pathophysiology, Helicobacter pylori, nitrite exposures, and pancreatic cancer. Studies in the literature provide a lot of suggestive evidence but are certainly not definitive for the hypotheses, and interpretations other than the ones I proposed are possible.

Capurso et al. . . . [Full Text of this Article]


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Related Article in JNCI

Etiology of Pancreatic Cancer, With a Hypothesis Concerning the Role of N-Nitroso Compounds and Excess Gastric Acidity
Harvey A. Risch
J Natl Cancer Inst 2003 95: 948-960. [Abstract] [Full Text] [PDF]

Related Correspondence

Re: Etiology of Pancreatic Cancer, With a Hypothesis Concerning the Role of N-Nitroso Compounds and Excess Gastric Acidity
Gabriele Capurso, Gianfranco Delle Fave, and Nick Lemoine
J Natl Cancer Inst 2004 96: 75. [Extract] [Full Text] [PDF]