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JNCI Journal of the National Cancer Institute 2002 94(23):1809; doi:10.1093/jnci/94.23.1809
© 2002 by Oxford University Press
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Journal of the National Cancer Institute, Vol. 94, No. 23, 1809, December 4, 2002
© 2002 Oxford University Press

CORRESPONDENCE

Re: Loss of Imprinting of Insulin-Like Growth Factor-II (IGF2) Gene in Distinguishing Specific Biologic Subtypes of Wilms Tumor

Ian M. Morison, David M.O. Becroft, Anthony E. Reeve

Affiliations of authors: I. M. Morison, A. E. Reeve, Cancer Genetics Laboratory, Department of Biochemistry, University of Otago, Dunedin, New Zealand; D. M. O. Becroft, Department of Obstetrics and Gynaecology, University of Auckland, Auckland, New Zealand.

Corresponding author: Ian M. Morison, M.B., Ch.B., Ph.D., Cancer Genetics Laboratory, Department of Biochemistry, University of Otago, P.O. Box 56, Dunedin, New Zealand (e-mail: ian.morison@otago.ac.nz).

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In a recent issue of the Journal, Ravenel et al. (1) claim that loss of imprinting (LOI) of the insulin-like growth factor-II (IGF2) gene defines a molecular subgroup of Wilms tumors that have a different pathologic subtype, a later age of onset, and greater IGF2 expression than those without LOI. On . . . [Full Text of this Article]


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