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JNCI Journal of the National Cancer Institute 2002 94(15):1174-1175; doi:10.1093/jnci/94.15.1174
© 2002 by Oxford University Press
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Journal of the National Cancer Institute, Vol. 94, No. 15, 1174-1175, August 7, 2002
© 2002 Oxford University Press

CORRESPONDENCE

RESPONSE: Re: Effect of Long-Term Estrogen Deprivation on Apoptotic Responses of Breast Cancer Cells to 17{beta}-Estradiol and The Two Faces of Janus: Sex Steroids as Mediators of Both Cell Proliferation and Cell Death

Ana M. Soto, Carlos Sonnenschein

Affiliations of authors: A. M. Soto, C. Sonnenschein, Department of Anatomy and Cellular Biology, Tufts University, Boston, MA.

Correspondence to: Ana M. Soto, M.D., Department of Anatomy and Cellular Biology, Tufts University School of Medicine, Boston, MA 02111 (e-mail: ana.soto@tufts.edu).

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The comments by Jordan et al. focus on yet another variation of the phenomenon of estrogen-induced tumor regression. The use of antiestrogens in the treatment of breast cancer was predicated on the idea that, if estrogens induce cancer growth, then antiestrogens should do the opposite. However, the clinical studies that resulted in the adoption of tamoxifen as the drug of choice in the treatment of estrogen receptor-positive breast . . . [Full Text of this Article]


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