Skip Navigation

JNCI Journal of the National Cancer Institute 2001 93(22):1671; doi:10.1093/jnci/93.22.1671
© 2001 by Oxford University Press
This Article
Right arrow Full Text Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Request Permissions
Google Scholar
Right arrow Search for Related Content

Journal of the National Cancer Institute, Vol. 93, No. 22, 1671, November 21, 2001
© 2001 Oxford University Press

IN THIS ISSUE

Estrogen-Deprived Breast Cancer Cells and Estradiol

Although estrogen can promote the growth of estrogen receptor-positive breast cancers, high doses of estrogen can have the paradoxical effect of promoting tumor regression in postmenopausal women. To investigate the mechanism of tumor regression, Song et al. (p. 1714) made use of LTED, E8CASS, and BSK3 cells, which are MCF-7 breast cancer cells grown for many months in the absence of estrogen. LTED cells treated with high doses of estradiol showed decreased cell growth and increased apoptosis. Identically treated MCF-7 cells, by contrast, grew at increased rates and showed reduced apoptosis. . . . [Full Text of this Article]

Trends in U.S. Cancer Screening

Interleukin 15 and Epstein-Barr Virus-Infected Lymphocytes

Incidence of Metachronous Testicular Cancer

Androgen-Independent Prostate Cancer and Dexamethasone

Measuring GSTP1 Methylation in Prostate Cancer