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JNCI Journal of the National Cancer Institute 2000 92(5):363-365; doi:10.1093/jnci/92.5.363
© 2000 by Oxford University Press
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Journal of the National Cancer Institute, Vol. 92, No. 5, 363-365, March 1, 2000
© 2000 Oxford University Press

EDITORIALS

Emerging Technologies and Cervical Cancer

Michele Follen, Rebecca Richards-Kortum

Affiliations of authors: M. Follen (formerly Michele Follen Mitchell), Department of Gynecologic Oncology, The University of Texas M. D. Anderson Cancer Center, Houston; R. Richards-Kortum, Department of Electrical and Computer Engineering, The University of Texas, Austin.

Correspondence to: Michele Follen, M.D., M.S., Department of Gynecologic Oncology, The University of Texas M. D. Anderson Cancer Center, 1515 Holcombe Blvd., Houston, TX 77030 (e-mail: mfollen@mdanderson.org).

Human papillomavirus (HPV) has been demonstrated to be a necessary cause of invasive cervical cancer across epidemiologic and virologic studies worldwide (1). However, the biologic mechanisms for malignant transformation by HPV were well established long before the epidemiologic studies showed any consistency among investigators or across study designs. Once sensitive biologic assays became available for the detection of HPV, correlational, case-control, and cohort studies demonstrated high relative risks, high attributable fractions, appropriate time sequence, differential risk among higher viral load and higher risk viral types, and consistency among investigators (2). Now, with the relationship of HPV to cervical cancer established, attention has turned to how to use this information to decrease the morbidity of and mortality from cervical cancer.

Cervical cancer is the third most common cancer in women worldwide. Parkin et al. (3) estimated that 371 200 cases of cervical cancer were diagnosed . . . [Full Text of this Article]

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