© 1999 by Oxford University Press
Journal of the National Cancer Institute, Vol. 91, No. 4, 299-301,
February 17, 1999
© 1999 Oxford University Press
EDITORIALS |
Molecular Detection of Early Lung Cancer
Affiliation of authors: Hamon Center for Therapeutic Oncology Research, University of Texas Southwestern Medical Center, Dallas.
Correspondence to: Adi F. Gazdar, M.D., Hamon Center for Therapeutic Oncology Research, University of Texas Southwestern Medical Center, 5323 Harry Hines Blvd., Dallas TX 75235-8593 (e-mail: gazdar@simmons.swmed.edu).
Lung cancer is the most common cause of cancer death in the United
States, and most cases follow long-term use of tobacco products
(1,2). The first objective in lung cancer control is to
prevent persons from starting to smoke and to help those who smoke to
quit. Nevertheless, in the United States today, there are 48 million
smokers, most of whom find it difficult to quit, with new smokers
rapidly replacing the quitters (3) and smokers who die from
their disease. In addition, about 40% of all new lung cancers are
occurring in former smokers, who remain at high risk for developing
lung cancer for many years after they stop smoking (4). Under
current standard methods of diagnosis and treatment, less than 15%
of patients with lung cancer will survive their disease (2).
Most patients who achieve long-term survival are
NOTES
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