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JNCI Journal of the National Cancer Institute 2008 100(3):157; doi:10.1093/jnci/djn008
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© Oxford University Press 2008.

IN THIS ISSUE

The first 10% of the full text of this article appears below.

Sorafenib Targets Mutant FLT3 in Acute Myelogenous Leukemia

The kinase inhibitor sorafenib induces growth arrest and apoptosis at much lower concentrations in acute myeloid leukemia (AML) cell lines that harbor internal tandem duplication (ITD) mutations in a section of the Fms-like tyrosine kinase 3 (FLT3) gene than in those with wild-type FLT3. Zhang et al. (p. 184) examined the anti-leukemic activity of sorafenib in mouse AML cell lines that expressed mutant or wild-type FLT3, human AML cells, a mouse leukemia xenograft model, and 16 AML patients with known FLT3 gene mutation status who were . . . [Full Text of this Article]

Endogenous Sex Hormones and Prostate Cancer Risk

Surgeon and Patient Attributes and Radiotherapy for Breast Cancer

Ki-67 Expression and Response to Breast Cancer Adjuvant Therapy

Personality Factors and Risk of Breast Cancer

ER Results from Pathology versus Central Laboratory


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